Tests in mice indicate that the bacterium that causes Lyme disease can commandeer a gene in its interim host—the deer tick—enabling the bacterium to escape immune detection once inside a mammal.
Researchers at Yale University report in the July 28 Nature that the Lyme microbe, Borrelia burgdorferi, activates a gene in the tick, boosting production of a salivary protein called Salp-15.
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